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Post by mominator on Nov 1, 2003 11:26:11 GMT -5
I was lurking at Bob's board and a particular post caught my attention.  Someone was questioning advice on this board regarding producing prolactin, Bob's response was producing prolactin is a temporary state, which is true...so I guess my question is, producing the prolactin is important and offer's temporary growth, but does it encourage fat cells to "stick" so to speak in the breast, therefore making for "actual" growth? Did that make sense?  |
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Post by mominator on Nov 1, 2003 11:26:59 GMT -5
Date Posted: 10/04/2003 2:42 PM
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Prolactin is necessary even if it is a temporary tissue gain. It is all temporary during growth stages. Anything can change it down the line.
I think that if one can get some growth out of prolactin raising practices then do it, and use the "other methods" to back fill it with permanent tissue.
my 2 cents
Wahaika
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Post by mominator on Nov 1, 2003 11:27:20 GMT -5
Date Posted: 10/08/2003 11:48 AM
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For what it's worth - my prolactin was VERY high I think when i started BE and massaging - my first post to this board was "uh, what's that white stuff coming out of my nipples!" And my breasts are tiiiny. So high prolactin by itself doesn't mean your breasts will grow fast.
aprilbabe
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Post by mominator on Nov 1, 2003 11:27:59 GMT -5
Date Posted: 10/09/2003 9:57 AM
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Okay - here's the history of my prolactin theory....by asking the following questions:
1. When does breast growth occur naturally? Puberty and during or post-pregnancy when nursing.
2. What hormones are elevated during these times and which ones are suppressed (or missing)?
Puberty: elevated estrogen & prolactin, missing - progesterone (during puberty girls rarely ovulate)
Post-pregnancy Nursing: elevated prolactin, missing progesterone.
Prolactin: Although it cycles slightly during the menstrual cycle, the only times it is elevated is during puberty and post pregnancy and through nursing.
Progesterone: Only produced in high levels during ovulation and by the placenta during pregnancy. During puberty, ovulation is very irregular, and sometime non-existant during the first year or two after menarche. During pregancy, ovulation is usually suppressed to some degree by nursing, and occurs infrequently or not at all during the nursing period.
The affects of prolaction on breast development are rather indirect. During puberty, it works with estrogen for the development of glandular tissue. Prolactin itself increases the amount of estrogen receptors in the mammary tissues (stroma, glands and fatty tissue), making them more receptive to estrogen.
It also decreases the amount of a enzyme called lipoprotein lipase (LPL) in all adipose tissue in the body - but NOT in the mammary glands. In this way, it works as a "targeting system". LPL is the enzyme that allows fat cells to remove tryglicerides from the blood stream and store them. LPL is the "fat storage" enzyme produced by the fat cell that helps them store fat. Prolactin decreases the amount of LPL produced by the fat cell - but it increases the amount produced in the mammary glands. The end result of this is that when increased estrogen levels (during a BE program) cause increased insulin response and subsequent fat storage, the reduced amount of LPL in adipose tissue will disable the storage of fat in the lower body, while increasing the uptake of nutrients in the mammary glands for growth. **
** Identification of functional prolactin (PRL) receptor gene expression: PRL inhibits lipoprotein lipase activity in human white adipose tissue., Ling C, Svensson L, Oden B, Weijdegard B, Eden B, Eden S, Billig H., Department of Physiology, Sahlgrenska University Hospital, Goteborg University, 405 30 Goteborg, Sweden.
**A review of the hormone prolactin during lactation., Ostrom KM., Department of Nutritional Sciences, University of Connecticut, Storrs.
As for progesterone, it has been shown that the glandular tissue grows the most during the luteal phase, when estrogen and progesterone are at a peak. This is glandular tissue, though, and not an increase in size, fatty or connective tissue. Progesterone blocks prolactin and cortosteroids, which are both responsible for anabolism (growth) of the fatty tissue.
The production of prolactin IS temporary - which means that after the first couple of days after birth, prolactin is ONLY produced in response to stimulation (nursing - or in our case, massage). For those of you who have nursed, you know what I mean. Part of weaning is reducing the frequency of nursing, putting more time between each nursing session. The production of prolactin is directly a result of stimulation - and stops after a while. Which is why I have always said that the frequency of the massage is more important than the length of the massage. A minute of massage every hour is more productive in terms of prolactin production than 24 minutes of massage once per day.
Combined with the affect of massage on circulation, an increase of prolactin, because of it's subtle affect on body fat distribution through the regulation of LPL, should aid a BE program.
So, that is my theory behind prolactin and how I achieved it:
1. Prolactin is only elevated during puberty and pregnancy/nursing - the two periods of natural breast growth.
2. Research into the affects of prolactin on fat storage and distribution.
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Fawn
Botanical Beauty Labs
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